Low-fat diets saw a real upswing in 1977. But according to research published in the Open Heart journal, there was no
scientific basis for the recommendations to cut fat from our diet in the first
place. What is worse, the
processed food industry replaced fat with large amounts of sugar (fructose). And this has
led to a massive increase in obesity, diabetes, heart disease and non-alcoholic
fatty liver disease. This is because unlike glucose, which can be used by
virtually every cell in our body, fructose can only be metabolized by liver. Since
nearly all fructose get shuttled to liver, it ends up taxing and damaging our liver in
the same way alcohol and other toxins do.
As the cholesterol hypothesis (we should not eat foods with high cholesterol content) is false, this also means that the recommended therapies—low-fat, low-cholesterol diet and cholesterol lowering medications—are doing more harm than good. Statin (a cholesterol-lowering drug) treatment, for example, is largely harmful, costly and has transformed millions of people into patients whose health is being adversely impacted by the drug.
As the cholesterol hypothesis (we should not eat foods with high cholesterol content) is false, this also means that the recommended therapies—low-fat, low-cholesterol diet and cholesterol lowering medications—are doing more harm than good. Statin (a cholesterol-lowering drug) treatment, for example, is largely harmful, costly and has transformed millions of people into patients whose health is being adversely impacted by the drug.
Part of
the reason why cholesterol-lowering drugs are
ineffective for heart disease prevention is that drugs cannot address the real
cause of heart disease, which is insulin and leptin resistance, which in
turn increase our LDL particle number.
While some genetic predisposition can play a role, insulin and leptin
resistance is primarily caused by a combination of factors that are epidemic in
our modern lifestyle:
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